Tohoku University · Chemical Engineering
Professor Hisashi Nakamura's research lab specializes in experimental and numerical studies of chemical kinetics, particularly focusing on ammonia oxidation at intermediate temperatures and in fuel-rich conditions. The lab employs advanced reactor systems such as micro flow reactors with precise temperature control to investigate fundamental reaction mechanisms. Additionally, the lab develops high-precision control systems, including innovative phase-locked loop-based servo controllers, for high-performance motion control in industrial applications like precision gear grinding. The integration of chemical kinetics and advanced mechatronics defines the lab’s interdisciplinary approach.
Figures are computed from collected data and may differ slightly.
In recent years, a number of experimental and numerical studies on chemical kinetics of ammonia (NH3) have been performed and NH3 chemical kinetic models have been proposed. However, as NH3 oxidation at intermediate temperatures (below 1400 K) has been investigated by reactor experiments, discrepancies between measurements and model-prediction have been reported. In addition, NH3 oxidation in extremely fuel-rich conditions has not been well studied. This study aims to further explore the chemica
This paper presents a new method proposed for high-precision positioning servomechanisms. The servo controller uses a two-phase-type phase-locked loop (PLL) to detect position tracking error and speed fluctuation with high resolution. Because the two-phase-type PLL has a wide frequency range and high noise suppression performance, we applied the new controller to high-power and high-velocity servo-spindles and achieved high control performance. The developed servo-spindles were used by a high-pr
These data demonstrate that Tenm4 mutants fail to form a primitive streak and to induce embryonic mesoderm. Markers of anterior posterior patterning fail to be expressed or are mislocalized. Further, Tenm4 mutants lack the ability to differentiate in a cell autonomous manner. Together, our data suggest that embryos become impaired prior to E6.5 and as a result, Wnt signaling fails to occur; however, the involvement of other signaling pathways remains to be examined.
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