Waseda University · 의학
다이비드 J. 스텐젤 교수의 연구실은 운동이 식이욕구와 에너지 섭취 조절에 미치는 영향을 생리학적·호르몬적 메커니즘으로 규명하는 데 초점을 맞추고 있습니다. 특히 허리프레드린, 페피티딘, 렙틴 등 식이욕구 조절 호르몬의 역할과 운동이 이들 호르몬에 미치는 영향을 중심으로 연구를 진행하고 있으며, 개인 간의 반응 차이를 고려한 개인 맞춤형 운동 및 식이 전략 개발을 목표로 하고 있습니다. 운동 후 일시적인 식욕 감소 메커니즘과 체중 조절의 생물학적 기초를 밝히는 데 기여하고 있습니다.
표시된 성과는 수집된 데이터 기준으로 산출되며, 일부 차이가 있을 수 있습니다.
Exercise facilitates weight control, partly through effects on appetite regulation. Single bouts of exercise induce a short-term energy deficit without stimulating compensatory effects on appetite, whilst limited evidence suggests that exercise training may modify subjective and homeostatic mediators of appetite in directions associated with enhanced meal-induced satiety. However, a large variability in responses exists between individuals. This article reviews the evidence relating to how adipo
Knowledge about the relationship between exercise and appetite is important both for athletes wishing to optimise performance and for those interested in maintaining a healthy body weight. A variety of hormones are involved in appetite regulation including both episodic hormones, which are responsive to episodes of feeding, and tonic hormones, which are important regulators of energy storage over the longer term (e.g. insulin and leptin). Notable among the episodic appetite-regulating hormones i
These findings suggest a mediating role of acylated ghrelin and PYY(3-36) in determining divergent feeding responses to energy deficits imposed by food restriction and exercise.
Ghrelin is a hormone that stimulates hunger. Intense exercise has been shown to temporarily suppress hunger after exercise. In the present study, we investigated whether post-exercise hunger suppression is mediated by reduced plasma total ghrelin concentrations. Nine men and nine women participated in the study. Their mean physical characteristics were as follows: age 24.8 (s(x) = 0.9) years, body mass index 22.9 (s(x) = 0.6) kg x m(-2), maximal oxygen uptake (VO(2max)) 57.7 (s(x) = 2.2) ml x kg
