Jongeun Choi
연세대학교 기계공학과 · 공학
Jongeun Choi 교수의 연구실은 스마트 센서 워크, 다중 에이전트 시스템, 그리고 생체 및 생체역학적 과정의 실시간 모니터링과 예측을 위한 지능형 알고리즘 개발에 중점을 두고 있습니다. 특히, 노이즈가 많은 측정값을 바탕으로 자기조직화되는 에이전트들이 공간-시간적 과정을 강인하게 추정하고 최적의 측정 전략을 수립하는 데에 초점을 맞추고 있습니다. 이는 의료 영상 분석, 골다공로시스 위험 평가, 복합동맥류의 생체역학적 진화 예측 등 실제 임상 및 의공학 응용과도 연결됩니다.
표시된 성과는 수집된 데이터 기준으로 산출되며, 일부 차이가 있을 수 있습니다.
This paper presents a novel class of self-organizing sensing agents that adaptively learn an anisotropic, spatio-temporal gaussian process using noisy measurements and move in order to improve the quality of the estimated covariance function. This approach is based on a class of anisotropic covariance functions of gaussian processes introduced to model a broad range of spatio-temporal physical phenomena. The covariance function is assumed to be unknown a priori. Hence, it is estimated by the max
The developed DL model significantly outperforms conventional ML models and clinical tools. Our XAI model produces high-ranked features along with the integrated contributions of each feature, which facilitates the interpretation of individual risk. In summary, our interpretable model for osteoporosis risk screening outperformed state-of-the-art methods.
This paper presents a novel class of self-organizing multi-agent systems that form a swarm and learn a spatio- temporal process through noisy measurements from neighbors for various global goals. The physical spatio-temporal process of interest is modeled by a spatio-temporal Gaussian process. Each agent maintains its own posterior predictive statistics of the Gaussian process based on measurements from neighbors. A set of biologically inspired navigation strategies are identified from the poste
Autophagy is known to be regulated by the phosphoinositide-3 kinase (PI3K)-protein kinase B (AKT) and/or mitogen-activated protein kinase 1/2 (MEK1/2)-extracellular signal-regulated kinase 1/2 (ERK1/2) pathways, leading to activation of mammalian target of rapamycin (mTOR), a major negative regulator of autophagy. However, some reports have also suggested that autophagic regulation by the PI3K-AKT and/or MEK1/2-ERK1/2 pathways may not be mediated by mTOR activity, and there is no direct evidence
Abdominal aortic aneurysms (AAAs) evolve over time, and the vertebral column, which acts as an external barrier, affects their biomechanical properties. Mechanical interaction between AAAs and the spine is believed to alter the geometry, wall stress distribution, and blood flow, although the degree of this interaction may depend on AAAs specific configurations. In this study, we use a growth and remodeling (G&R) model, which is able to trace alterations of the geometry, thus allowing us to compu
