東北大学 · Engineering
마사루 와타나베 교수의 연구실은 초임계 및 초임계 유체에서의 화학 반응, 특히 당류의 탈수 반응을 통한 유용한 화학물질(예: 5-HMF) 합성에 초점을 맞추고 있습니다. 고온 고압 조건에서의 촉매 반응 메커니즘과 반응 조건 최적화를 통해 효율적이고 지속 가능한 화학 공정을 개발하고 있으며, 마이크로파 가열 기술의 응용도 활발히 연구하고 있습니다. 또한 식물 발달 생물학 분야에서 아랍시드스의 표피 형성에 관여하는 수용체 히스티딘 키나제 유전자 기능을 규명하는 연구도 수행하고 있습니다.
표시된 성과는 수집된 데이터 기준으로 산출되며, 일부 차이가 있을 수 있습니다.
Catalytic dehydration of fructose into 5-hydroxymethylfurfural by microwave heating was studied in acetone-water mixtures in the presence of a cation exchange resin catalyst. The use of acetone–water reaction media resulted in yields of 5-HMF as high as 73.4% for 94% conversion at 150 °C. It was confirmed that there was no decrease of catalytic activity and selectivity for five reuses of the resin, which was in accordance with the elemental analysis results that showed that sulfonic acid groups
ADVERTISEMENT RETURN TO ISSUEPREVReviewNEXTChemical Reactions of C1 Compounds in Near-Critical and Supercritical WaterMasaru Watanabe*†, Takafumi Sato‡, Hiroshi Inomata*†, Richard Lee Smith Jr.†, Kunio Arai Jr.†, Andrea Kruse*§, and Eckhard Dinjus§View Author Information Research Center of Supercritical Fluid Technology, Tohoku University, 6-6-07 Aoba, Aramaki, Aoba-ku, Sendai 980-8579, Japan, Department of Applied Chemistry, Utsunomiya University, 7-1-2 Yoto, Utsunomiya 321-8585, Japan, and Ins
The ultra-sensitive ddPCR assay revealed that pretreatment T790M was found in the majority of NSCLC patients with EGFR-activating mutations. ddPCR should be utilized for detailed assessment of the impact of the low frequency pretreatment T790M mutation on treatment with EGFR-TKIs.
In higher plants, an outer layer of meristematic cells, the protoderm, forms early in embryogenesis and this layer gives rise to the epidermis in differentiating tissues. We proposed previously that an Arabidopsis thaliana homolog of crinkly4 (ACR4), a gene for a receptor-like protein kinase, would be involved in differentiation and/or maintenance of epidermis-related tissues. In the present study, we isolated loss-of-function acr4 mutants by a reverse genetic approach. Our extensive analyses us
