Myung‐Joo Kim
서울대학교 치의학과 · 치의학
김명주 교수의 연구실은 치과 및 의료 분야에서의 생체재료 개발과 임상 적용을 중심으로 연구를 진행하고 있습니다. 고분자 기반 생체재료인 PEEK, 티타늄 합금의 표면 특성 제어, 그리고 3D 프린팅 기반 치과 보철물의 정밀도 향상에 초점을 맞추고 있으며, 특히 생체적합성, 기계적 내구성, 조직 반응 최적화를 목표로 하고 있습니다. 최근에는 레이저 조각 기반 제조 기술과 생물학적 반응 분석을 융합한 정밀의료 기기 개발에도 기여하고 있습니다.
표시된 성과는 수집된 데이터 기준으로 산출되며, 일부 차이가 있을 수 있습니다.
Abstract The poly‐ether‐ether‐ketone (PEEK) polymer is of great interest as an alternative to titanium in orthopedics because of its biocompatibility and low elastic modulus. This study evaluated the fatigue limits of PEEK and the effects of the low elastic modulus PEEK in relation to existing dental implants. Compressive loading tests were performed with glass fiber‐reinforced PEEK (GFR‐PEEK), carbon fiber‐reinforced PEEK (CFR‐PEEK), and titanium rods. Among these tests, GFR‐PEEK fatigue tests
The purpose of this study was to define the surface properties of prepared titanium (Ti) disks, which served as a model system, and to contrast the biologic response of MG63 cells exposed to Ti disks with different levels of surface roughness. The surface properties interact with each other, resulting in a change of other surface qualities in addition to roughness due to the surface roughening procedure. The machined Ti disks were roughened by sandblasting and electric glow discharging. The surf
Laser sintered technology has been introduced for clinical use and can be utilized more widely, accompanied by the digitalization of dentistry and the development of direct oral scanning devices. This study was performed with the aim of comparing the marginal accuracy and internal fit of Co-Cr alloy copings fabricated by casting, CAD/CAM (Computer-aided design/Computer-assisted manufacture) milled, and 3-D laser sintered techniques. A total of 36 Co-Cr alloy crown-copings were fabricated from an
Laparoscopic cornual resection is a feasible primary approach for the management of heterotopic cornual pregnancy.
We compared the effects of ethylacetate extracts from Artemisia iwayomogi (AIE) and Artemisia capillaris (ACE) on ethanol-induced hepatic injury in mice. Ethanol (25% v/v, 5 g/kg body weight) was orally administered once a day for 6 wk. AIE or ACE was provided in the diet (0.05 g/100 g diet). AIE and ACE did not affect hepatic alcohol dehydrogenase activity but did significantly inhibit cytochrome P450 2E1 activity. Hepatic acetaldehyde dehydrogenase 2 activity significantly increased in the AIE
