Ulsan National Institute of Science and Technology · 工学
Professor Jaeseon Lee's research lab specializes in advanced thermal management for high-power-density electronics, with a primary focus on innovative cooling technologies to address extreme heat dissipation challenges in defense and high-performance computing applications. The lab investigates microchannel heat sinks integrated with refrigeration cycles—particularly R134a-based systems—enabling precise temperature control below 55°C even at heat fluxes exceeding 100 W/cm². Research also emphasizes the mitigation of system inefficiencies such as wet compression and liquid entrainment, aiming to enhance reliability and performance of direct and indirect refrigeration cooling systems. The lab’s work bridges fundamental heat transfer principles with practical engineering solutions for next-generation electronic systems.
Figures are computed from collected data and may differ slightly.
<para xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink"> For a given heat sink thermal resistance and ambient temperature, the temperature of an electronic device rises fairly linearly with increasing device heat flux. This relationship is especially problematic for defense electronics, where heat dissipation is projected to exceed 1000 <formula formulatype="inline"> <tex Notation="TeX">${\hbox {W}}/{\hbox {cm}}^{2}$</tex></formula> in the near future. Dir
JAK inhibition controls aberrant activation of SGECs and may be a novel therapeutic approach for primary SS.
C57BL/6 mice are known to be resistant to the development of collagen-induced arthritis (CIA). However, they show a severe arthritic phenotype when the Ifng gene is deleted. Although it has been proposed that IFN-γ suppresses inflammation in CIA via suppressing Th17 which is involved in the pathogenesis of CIA, the exact molecular mechanism of the Th17 regulation by IFN-γ is poorly understood. This study was conducted to 1) clarify that arthritogenic condition of IFN-γ knockout (KO) mice is depe
Objective Baricitinib, a selective inhibitor for janus kinase (JAK) 1 and JAK2, is approved for use in rheumatoid arthritis. Systemic lupus erythematosus (SLE) is recently regarded as a potential candidate targeted by JAK inhibitors because of the relationship between its pathogenesis and JAK/signal transducer and activator of transcription (STAT) pathway-mediated cytokines such as type I interferons. The objective of this study was to determine whether baricitinib could effectively ameliorate S
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