이정미 교수

This paper explores the preliminary process reading that arises from achievements in the progressive. The meaning of the progressive as a partitive operator poses challenges in the formalization of its occurrence with punctual predicates like achievements. I discuss problems with prior proposals (Rothstein 2004, Bohnemeyer 2005), and argue that the preliminary process reading should be accounted for in terms of the meaning of achievements rather than the meaning of the progressive. Based on Bach

This paper examines the effect of lexical aspect on the evidential reading of a Korean sentence with the evidential marker -te. I show that statives and inchoatives exhibit different patterns of interaction with tense in a -te sentence. I analyze both cases in line with Lee (2011a, 2013), and account for how evidential readings arise from relevant components in the proposed analysis. On the basis of the compositional analysis, I argue that the generalization on the interaction between tense and

This paper explores the difference of status of the two implications that arise from Korean evidential sentences, and their interactions in the course of interpretation. The research shows that at-issue content and not-at-issue content interact bi-directionally in the temporal and modal interpretation of an evidential sentence. As such, the empirical patterns observed in Korean evidential sentences pose a problem for the formal systems that have been proposed by Faller (2002) and Potts (2005), a

This study presents a unified account of epistemic bias in Korean Negative Interrogatives by redefining contextual evidence bias as addressee bias. The empirical findings show that bias polarity and orientation are jointly determined by two factors: the interpretational status of negation and the prosodic contour of rising intonation. Truth-functional negation with unmarked prosody yields a negative, addressee-anchored bias, whereas non-truth-functional negation with marked prosody produces a po

CD137 (4-1BB/tnfrsf9) has been shown to co-stimulate T cells. However, agonistic anti-CD137 monoclonal antibody (mAb) treatment can suppress CD4＋ T cells, ameliorating autoimmune diseases, whereas it induces activation of CD8＋ T cells, resulting in diverse therapeutic activity in cancer,viral infection. To investigate the CD137-mediated T cell suppression mechanism, we examined whether anti-CD137mAb treatment could affect CD11b＋Gr-1＋ myeloid-derived suppressor cells (MDSCs). Intriguingly, anti-C