Nam-sik Han
Yonsei University
研究室紹介
Professor Nam-sik Han's research lab focuses on understanding the complex interplay between psychological factors such as social anxiety, peer relationships, and social support in adolescents, with a particular emphasis on bullying dynamics and mental health. The lab also engages in molecular and genetic research, especially in cancer biology, where it investigates therapeutic strategies for diseases like chronic myeloid leukemia through network-based simulations and targeted gene delivery using chimeric nanoparticles. The integration of behavioral science and molecular therapeutics defines the lab’s interdisciplinary approach.
Research Overview
Research Output Trend
Figures are computed from collected data and may differ slightly.
Selected Papers
7The purpose of this study was to investigate the ways in which relations among Korean middle-school students' interpersonal relationships, peer bullying, and social support affected the degree of social anxiety they experienced. The subjects were 594 adolescents (288 males, 306 females) recruited from five middle schools located in Seoul. The instruments used in the study included the Social Anxiety Scale(Moon, & Oh, 2002), the Revised Adult Attachment Scale(Collins, & Read, 1990), Bully/Victim
Supplementary Material 3 Literature gene and the corresponding papers
Supplementary Material 2
Abstract Developing an efficient and safe therapy necessitates a mechanistic understanding of the complex underlying pathology and manipulation of the multiple pathways at the molecular and genetic level. Network-based simulation of chronic myeloid leukemia (CML), a relatively well-understood cancer model, revealed the dynamics of simultaneously expressing pro-apoptotic BIM (Bcl-2 interacting mediator of cell death) and silencing pro-survival MCL-1 (myeloid cell leukemia-1) in combination with t
Supplementary Material 2
Supplementary Material 3 Literature gene and the corresponding papers
Abstract Developing an efficient and safe therapy necessitates a mechanistic understanding of the complex underlying pathology and manipulation of the multiple pathways at the molecular and genetic level. Network-based simulation of chronic myeloid leukemia (CML), a relatively well-understood cancer model, revealed the dynamics of simultaneously expressing pro-apoptotic BIM (Bcl-2 interacting mediator of cell death) and silencing pro-survival MCL-1 (myeloid cell leukemia-1) in combination with t