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한남식 교수

Nam-sik Han

연세대학교

연구실 소개

한남식 교수의 연구실은 청소년기 사회적 불안, 동료 갈등, 지원 체계 간의 상호작용을 심리학적·사회적 관점에서 탐구하는 데 초점을 맞추고 있습니다. 특히 중학생을 대상으로 한 bullying, 애자기 관계, 사회적 지지가 정서적 불안에 미치는 영향을 다각도로 분석하며, 정서적 건강 증진을 위한 실질적 개입 전략 개발을 목표로 합니다. 이와 더불어 분자생물학적 기반의 질병 메커니즘 연구를 통해 암 치료의 효율성과 안전성을 높이기 위한 유전자 및 단백질 네트워크의 시뮬레이션 기반 연구도 병행하고 있습니다.

사회적 불안청소년 심리폭력 예방유전자 네트워크항암 치료

연구 현황

논문 수
7
총 인용 수
16
최근 5년 논문
7
주요 분야

연구 성과 추이

표시된 성과는 수집된 데이터 기준으로 산출되며, 일부 차이가 있을 수 있습니다.

5개년 연도별 논문 게재 수
7총합
2012
2026
5개년 연도별 피인용 수
16총합
20122026

주요 논문

7
1
논문|인용수 16·2012
중학생의 대인관계 성향, 또래 괴롭힘, 사회적 지지와사회적 불안과의 관계
한남식, 어주경

The purpose of this study was to investigate the ways in which relations among Korean middle-school students' interpersonal relationships, peer bullying, and social support affected the degree of social anxiety they experienced. The subjects were 594 adolescents (288 males, 306 females) recruited from five middle schools located in Seoul. The instruments used in the study included the Social Anxiety Scale(Moon, & Oh, 2002), the Revised Adult Attachment Scale(Collins, & Read, 1990), Bully/Victim

2
dataset|인용수 0·2026
Additional file 3 of Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation
Margaret L. Lugin, Winnie Lei, Rebecca T. Lee, Jee Young Chung, Nicholas M. Katritsis, Woochang Hwang, Angela Fleischman, Namshik Han, Young Jik Kwon
FigshareOA

Supplementary Material 3 Literature gene and the corresponding papers

3
dataset|인용수 0·2026
Additional file 2 of Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation
Margaret L. Lugin, Winnie Lei, Rebecca T. Lee, Jee Young Chung, Nicholas M. Katritsis, Woochang Hwang, Angela Fleischman, Namshik Han, Young Jik Kwon
FigshareOA

Supplementary Material 2

4
other|인용수 0·2026
Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation
Margaret L. Lugin, Winnie Lei, Rebecca T. Lee, Jee Young Chung, Nicholas M. Katritsis, Woochang Hwang, Angela Fleischman, Namshik Han, Young Jik Kwon
FigshareOA

Abstract Developing an efficient and safe therapy necessitates a mechanistic understanding of the complex underlying pathology and manipulation of the multiple pathways at the molecular and genetic level. Network-based simulation of chronic myeloid leukemia (CML), a relatively well-understood cancer model, revealed the dynamics of simultaneously expressing pro-apoptotic BIM (Bcl-2 interacting mediator of cell death) and silencing pro-survival MCL-1 (myeloid cell leukemia-1) in combination with t

5
dataset|인용수 0·2026
Additional file 2 of Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation
Margaret L. Lugin, Winnie Lei, Rebecca T. Lee, Jee Young Chung, Nicholas M. Katritsis, Woochang Hwang, Angela Fleischman, Namshik Han, Young Jik Kwon
FigshareOA

Supplementary Material 2

6
dataset|인용수 0·2026
Additional file 3 of Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation
Margaret L. Lugin, Winnie Lei, Rebecca T. Lee, Jee Young Chung, Nicholas M. Katritsis, Woochang Hwang, Angela Fleischman, Namshik Han, Young Jik Kwon
FigshareOA

Supplementary Material 3 Literature gene and the corresponding papers

7
other|인용수 0·2026
Multimodal gene and targeted drug therapy for chronic myelogenous leukemia using BIM/MCL-1 chimeric nanoparticle: computational target analysis and therapeutic validation
Margaret L. Lugin, Winnie Lei, Rebecca T. Lee, Jee Young Chung, Nicholas M. Katritsis, Woochang Hwang, Angela Fleischman, Namshik Han, Young Jik Kwon
FigshareOA

Abstract Developing an efficient and safe therapy necessitates a mechanistic understanding of the complex underlying pathology and manipulation of the multiple pathways at the molecular and genetic level. Network-based simulation of chronic myeloid leukemia (CML), a relatively well-understood cancer model, revealed the dynamics of simultaneously expressing pro-apoptotic BIM (Bcl-2 interacting mediator of cell death) and silencing pro-survival MCL-1 (myeloid cell leukemia-1) in combination with t

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